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Role of MEK, PI3, p38, Tyrosine, and mTOR Kinases in Regulation of Heart Resistance to the Arrhythmogenic Action of Short-Term Ischemia and Reperfusion

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MEK, PI3, p38, tyrosine, and mTOR kinases are not involved in the regulation of heart resistance to the arrhythmogenic action of short-term ischemia/reperfusion in non-adapted rats.

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Authors and Affiliations

  1. Research Institute of Cardiology, Tomsk, Russia

    L. N. Maslov, A. V. Mukhomedzyanov, S. Yu. Tsibulnikov, E. I. Chauski, I. G. Khaliulin & A. G. Portnichenko

Authors
  1. L. N. Maslov
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  2. A. V. Mukhomedzyanov
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  3. S. Yu. Tsibulnikov
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  4. E. I. Chauski
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  5. I. G. Khaliulin
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  6. A. G. Portnichenko
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Correspondence to L. N. Maslov.

Additional information

Translated from Byulleten’ Eksperimental’noi Biologii i Meditsiny, Vol. 158, No. 12, pp. 688-691, December, 2014

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Maslov, L.N., Mukhomedzyanov, A.V., Tsibulnikov, S.Y. et al. Role of MEK, PI3, p38, Tyrosine, and mTOR Kinases in Regulation of Heart Resistance to the Arrhythmogenic Action of Short-Term Ischemia and Reperfusion. Bull Exp Biol Med 158, 729–731 (2015). https://doi.org/10.1007/s10517-015-2848-2

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  • DOI: https://doi.org/10.1007/s10517-015-2848-2

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