Correcting Acne Scars Using PCI

Abstract

The deeper the scar, the greater the challenge. Scars in which the epidermis has been completely destroyed with melanin loss and result in atrophic scarring are also more difficult to treat. The lesions on the face are more responsive to PCI when compared to those found on the chest or back; the latter need more interventions to offer the same result as the former. Scars located in more oily areas, in the author’s experience, offer a better response to treatment, when compared to those arranged in regions with fewer seborrheic glands. This characterizes a differential of PCI, a technique that only needle the skin, in relation to the incision. In the latter, the enlargement and often the worsening of the scar is evidenced, most commonly in oily skin. Cystic acne usually results in scars that are difficult to treat. The consumption of the dermis and hypodermis, as well as the deterioration of the epidermis resulting from the destructive action of inflammatory cytokines, gives rise to depressed, elevated, dystrophic lesions, in addition to loss of pigment, hyperpigmentation, flaccidity, and development of superficial and deep rhytides. This polymorphism, commonly observed in patients after severe and prolonged inflammatory acne, presents itself as a therapeutic challenge. For this reason, it is necessary to evaluate these lesions in a particular way, examining their architecture and directing the option of more specific intervention to the correction of each cicatricial unit.