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The Impact of Infant Feeding on Later Metabolic Health

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Nutrition in Infancy

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Abstract

The objective of this chapter is to summarize the recent research related to the effect of infant feeding practices on the development of metabolic risk factors, overweight and obesity in early and later childhood. The Barker hypothesis provides a link between antenatal nutrition, postnatal growth, and subsequent adult disease [1, 2]. In fact, events in early life may program the function of a number of organ systems [2]. The fetal origins hypothesis states that fetal undernutrition in middle to late gestation leads to disproportionate fetal growth programming later coronary heart disease [3]. One public health strategy to challenge this hypothesis is the promotion of breastfeeding of all infants because breastfeeding may attenuate subsequent programming effects. Breastfeeding has been shown to protect against child obesity and cardiovascular risk outcomes [4], and is “dose related”; the longer an infant is breastfed, the lower the risk of obesity [5].

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Acknowledgment

WH Oddy is supported by a National Health and Medical Research Council of Australia Population Health Fellowship. Work for this chapter conducted by Ms McHugh was funded from a National Heart Foundation/Beyond Blue Strategic Research Initiative.

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Correspondence to Wendy Oddy .

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Oddy, W., McHugh, M.F. (2013). The Impact of Infant Feeding on Later Metabolic Health. In: Watson, R., Grimble, G., Preedy, V., Zibadi, S. (eds) Nutrition in Infancy. Nutrition and Health. Humana Press, Totowa, NJ. https://doi.org/10.1007/978-1-62703-224-7_15

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