- Elliot J. RothAffiliated withFeinberg School of Medicine Physical Medicine and Rehabilitation, Northwestern University
Hemorrhagic bleeding into the thalamus, typically resulting from hypertension.
Thalamic hemorrhage causes a variety of neurological findings, most commonly contralateral hemianesthesia (numbness), mild hemiparesis, hemiataxia, and sometimes intolerable intractable severe dysesthetic pain in half of the body (previously called “Dejerine–Roussy syndrome” or “thalamic pain,” but now called “central poststroke pain”). Because the thalamus is involved, sensory symptoms tend to predominate. Onset is usually during activity. Specific symptoms depend on the size and specific location of the hemorrhage, but can include stupor, coma, and signs of elevated intracranial pressure in their most severe forms. Virtually, any type of focal neurological change can be seen. Interestingly, while the motor and sensory deficits noted above are most common, oculomotor findings occur and even mild aphasia, cognitive deficits, and neurobehavioral dysfunction ...
Reference Work Entry Metrics
- Thalamic Hemorrhage
- Reference Work Title
- Encyclopedia of Clinical Neuropsychology
- pp 2504-2505
- Print ISBN
- Online ISBN
- Springer New York
- Copyright Holder
- Springer Science+Business Media, LLC
- Additional Links
- Industry Sectors
- Editor Affiliations
- 671. Physical Medicine and Rehabilitation, and Professor of Neurosurgery, and Psychiatry Virginia Commonwealth University – Medical Center Department of Physical Medicine and Rehabilitation
- 672. Kessler Foundation Research Center
- 673. Professor of Physical Medicine and Rehabilitation, and Neurology and Neuroscience, University of Medicine and Dentistry of New Jersey – New Jersey Medical School
- 674. Independent Practice
- Elliot J. Roth (2055)
- Author Affiliations
- 2055. Feinberg School of Medicine Physical Medicine and Rehabilitation, Northwestern University, 345 E. Superior, 60611, Chicago, IL, USA
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