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Pathophysiology of Retinal Vein Occlusions

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Retinal Vein Occlusions

Abstract

The pathophysiology of retinal vein occlusion (RVO) consists of the three components of Virchow’s triad – abnormalities of the vessel wall (considered in Chap. 1), alterations in the blood (e.g., abnormalities of viscosity and coagulation), and alterations in blood flow.1 This chapter addresses alterations of the blood and of blood flow.

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Notes

  1. 1.

    The astute reader will question how a typical critical closing value can be approximately 70 mmHg and yet approximately 7 mmHg less than SBP. SBP is usually quite a bit higher than 77 mmHg. The only thing I can think of is that the scleral suction cup gave the experimental subjects a bit of a vagal reaction. In any case, those numbers are what the paper states.

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Browning, D.J. (2012). Pathophysiology of Retinal Vein Occlusions. In: Retinal Vein Occlusions. Springer, New York, NY. https://doi.org/10.1007/978-1-4614-3439-9_2

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