Translational Stroke Research
Translational Stroke Research covers basic, translational, and clinical studies. The Journal emphasizes novel approaches in order to help translate scientific discoveries from basic stroke research into the development of new strategies for prevention, assessment, treatment, and repair after stroke and other forms of neurotrauma.
Translational Stroke Research focuses on translational research and is relevant to both basic scientists and physicians, including but not restricted to neuroscientists, vascular biologists, neurologists, neuroimagers, and neurosurgeons. The Journal provides an interactive forum for the dissemination of original research articles, review articles, research reports, letters, comments, and research protocols, in stroke and stroke related areas. Its distinguished editorial board is made up of leading stroke researchers and physicians from North America, Europe, and Asia.
CALL FOR PAPERS: "Phenotypic Transformation of Smooth Muscle in Cerebrovascular Disease"
Guest editor: William Pearce (Loma Linda University)
SUBMISSION DEADLINE: September 31, 2013
Smooth muscle can differentiate into multiple stable phenotypes that respond dynamically to physiological and pathophysiological influences. Whereas the phenotypic transformation of smooth muscle is well recognized in the context of arteriosclerosis, it may also contribute to the development of cerebral aneurisms, Alzheimer’s disease, brain trauma, stroke, and intracranial hemorrhage. The goal of this special issue is to review exciting recent advances in understanding of the role of phenotypic transformation of vascular smooth muscle in the development of cerebrovascular disease.
CALL FOR PAPERS: "Ion Transporters and Glutamate Receptor-independent Mechanisms for Ischemic and/or Traumatic Brain Injury"
Guest editor: Dandan Sun (University of Pittsburgh), Kristopher Kahle (Massachusetts General Hospital and Harvard Medical School
SUBMISSION DEADLINE: July 31, 2013
Glutamate receptor-mediated ionic imbalance and neurotoxicity have been investigated intensively for stroke and other CNS disorders. Emerging recent studies suggest that non-NMDA receptor-dependent mechanisms play an important role in regulation of ionic homeostasis in the CNS in ischemic and traumatic injury, including acid sensing ion channels, TRP channels, Cl- transporters, Na+/Ca2+ exchangers, and Na+/H+ exchangers. The purpose of this special issue is to present original studies and/or reviews on these newly emerging mechanisms of ionic dysregulation in the field.
CALL FOR PAPERS: “Protein Misfolding and Organell Stress after Brain Ischemia”
Guest editors: Donald DeGracia (Wayne State University) and Bingren Hu (University of Maryland)
SUBMISSION DEADLINE: August 31, 2013
Protein misfolding is an important mechanism for neurodegenerative diseases. Recent studies provide strong evidence that brain ischemia also leads to primary protein misfolding in organelles, and thus elicits organelle stress including heat shock, protein translational arrest, ER stress and other stress response programs. The objective of this special issue is to collect the most updated information about protein misfolding, organelle damage and stress responses after brain ischemia.
- 6 Volumes
- 26 Issues
- 379 Articles
- 34 Open Access
- 2010 - 2015 Available between
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