Abstract
Due to slight injury, the weakened vessel wall ruptures, producing an extravasation of blood, in which the tubercle bacilli settle down. Eventually this forms a tubercle follicle along with endothelial cells, lymphocytes, and giant cells. Gradually this follicle enlarges and is seen to the naked eye as a nodule in the bone marrow. Certain changes occur in the tuberculous nodule which are as follows.
Access this chapter
Tax calculation will be finalised at checkout
Purchases are for personal use only
References
Hodgson AR, Yau A, Kwon JS, Kim D. A clinical study of one hundred consecutive cases of Pott’s paraplegia. Clin Orthop. 1964;36:128–50.
Hodgson AR, Skingnes OK, Leong CY. Pathogenesis of Pott’s paraplegia. J Bone Joint Surg Am. 1967;49A:1147–56.
Girdlestone GR. Tuberculosis of bone and joint. London: Oxford University Press; 1940.
Griffiths DL, Seddon HJ, Roaf R. Pott’s paraplegia. London: Oxford University Press; 1956.
Author information
Authors and Affiliations
Corresponding author
Editor information
Editors and Affiliations
Rights and permissions
Copyright information
© 2013 Springer-Verlag London
About this chapter
Cite this chapter
Iyer, K.M. (2013). Tuberculosis. In: Iyer, K. (eds) General Principles of Orthopedics and Trauma. Springer, London. https://doi.org/10.1007/978-1-4471-4444-1_7
Download citation
DOI: https://doi.org/10.1007/978-1-4471-4444-1_7
Published:
Publisher Name: Springer, London
Print ISBN: 978-1-4471-4443-4
Online ISBN: 978-1-4471-4444-1
eBook Packages: MedicineMedicine (R0)