Abstract
I want to make it clear that this paper deals with some forms of squint where certain movements are limited but not because of any ocular muscle paralysis. I have been asked to include specifically dysthyroid ophthalmopathy in this paper and not all ophthalmologists would include such cases under the term ‘paralytic’ strabismus. Reduced ocular rotation may result from decreased innervation input to a muscle or failure of its antagonist to relax and extend normally. Furthermore, changes in orbital connective tissue may prevent normal movement of the eye despite the muscles contracting powerfully.
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© 1982 Dr W. Junk Publishers, The Hague
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Fells, P. (1982). The Treatment of Non-Comitant Strabismus. In: van Balen, A.T.M., Houtman, W.A. (eds) Strabismus Symposium Amsterdam, September 3–4, 1981. Documenta Ophthalmologica Proceedings Series, vol 32. Springer, Dordrecht. https://doi.org/10.1007/978-94-009-7997-0_30
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DOI: https://doi.org/10.1007/978-94-009-7997-0_30
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