Re-estimating the Demographic Impact on Health Care Expenditure: Evidence from Taiwan
- First Online:
DOI: 10.1057/gpp.2008.26
- Cite this article as:
- Shiu, YM. & Chiu, MC. Geneva Pap Risk Insur Issues Pract (2008) 33: 728. doi:10.1057/gpp.2008.26
Abstract
Unlike prior studies, we consider not only population ageing but also life expectancy as demographic variables that may explain the variations in health care expenditure. Cointegration techniques are employed to re-examine the effect of demographic changes on the Taiwan health care expenditure from 1960 to 2006. We find that the Taiwan health care expenditure, income, population ageing, life expectancy and the number of practicing physicians have statistically significant and long-run economic relationships. All variables have a positive impact on health care expenditure besides life expenditure. Our empirical results support the compression of morbidity hypothesis, suggesting that older people become healthier than in the past and that health care expenditure would decrease with life expectancy. Several implications for policymaking and future research are offered.
Keywords
health care expenditure population demographic impact population ageing life expectancy longevity compression of morbidityIntroduction
Over the past decades, most developed countries have experienced significant changes in their population structure, with ageing perhaps being the most important change.^{1} There are two possible reasons for this, one is the decline in the birth rate and the other is the natural extension of life expectancy (denoted as LE). Health policymakers are concerned that the increasing numbers of older people will increase the need for health expenditure and most people intuitively believe that such expenditure rises along with LE. Longevity has thus becomes an important issue with regard to health care spending.
In recent years, many studies have extensively examined the determinants of health care expenditure (denoted as HCE) by using macro- or micro-data. At the aggregated level, most research shows that age structure or population ageing has a small or non-significant impact on HCE.^{2} At the individual level, some authors argue that population ageing is a red herring that does not deserve the attention it usually receives. They find that population ageing is not the primary driver of HCE,^{3} highlighting instead the important effects of time to death or health status. Notably, when these studies add the time to death proxy to the account, the influence of age on HCE is significantly reduced.
Many previous studies state that the income proxy has a more significant impact than age structure.^{4} However, several investigations still find that there is a significant and positive correlation between the proportion of elderly people and HCE.^{5} In summary, it is difficult to come to any conclusions about the impact of demographics on HCE due to the discordant opinions on population ageing. However, since the LE change would influence the change in health status for aged groups, we add this factor to the HCE model to estimate the complete impact of demographics.
The structure of health care demand is likely to change due to increases in LE and falls in mortality. There are two opposite hypotheses on the impacts of the increase in LE on health care cost with constant medical technology. First is the expansion of the morbidity hypothesis, which assumes that the main effect of medical progress is to prolong the life of those patients who are so sick that they spend most of the additional years in the sickbed. Another is the compression of morbidity hypothesis, which states that an increase in LE, through a drop in the total mortality rate, will even lower per capita HCE.^{6} Most empirical evidence supports the compression of morbidity. Dormont et al.^{7} find that changes in morbidity induce health improvements that more than offset the increase in spending due to population ageing, meaning that longer lives do not necessarily entail rising HCE, and some empirical studies even find that an increase in longevity actually slows the growth in spending.^{8} Furthermore, Michel and Robine^{9} propose a new theory of the ageing population based on a cyclical movement. At first, sicker people survive into old age and disability rises, and then the number of years lived with disability decreases as new cohorts of healthier people enter old age. Finally, the number of years lived with disability increases again when the average age of death rises so much that many people spend their last years at an advanced age burdened by multiple chronic diseases and frailty. Thus co-exist the extension and compression of morbidity and this means that although people live longer it does not entail increasing HCE.
We employ cointegration techniques to examine the long-run economic equilibrium between demographic factors, non-demographic factors and HCE. Our empirical results show that HCE, income, population ageing, LE, and the number of practicing physicians have a statistically significant relationship in the long run. We find that LE from aged groups is significantly and negatively related to HCE. The result is consistent with the time-to-death hypothesis, suggesting that HCEs increase significantly as death approaches and decrease when lifespan extends, and that the effect of age is greatly reduced when adding the time to death factor.^{12} Our findings strongly support the compression of morbidity, which assumes that older people have longer LE and improved health status. When these elderly reach the natural limits of longevity, their period of sickness gets compressed into a shorter period before death.^{13} We reason that the lengthening of LE can possibly reduce the growth in HCE. This finding is obviously of significance for health service policymakers and financial planners.
The paper is organised as follows. In the next section, we present the hypotheses concerning the impact of population ageing and LE on HCE. The subsequent section describes our data and methodology. In the penultimate section, we discuss the empirical results. The conclusions and policy implications are provided at the end of this paper.
Population ageing, life expectancy and health care expenditure
Population and health care expenditure by three special age groups
Rates of HCE (%) | Rates of population (%) | Per capita HCE (NT$) | |||||||
---|---|---|---|---|---|---|---|---|---|
Age groups | 0–14 | 15–64 | 65+ | 0–14 | 15–64 | 65+ | 0–14 | 15–64 | 65+ |
1998 | 13.46 | 64.12 | 26.77 | 21.96 | 69.79 | 8.26 | 8,045 | 13,194 | 44,024 |
2002 | 12.22 | 63.06 | 29.40 | 20.42 | 70.56 | 9.02 | 8,990 | 14,181 | 51,822 |
2006 | 9.60 | 60.66 | 33.32 | 18.12 | 71.88 | 10.00 | 10,357 | 16,773 | 69,357 |
LE is a statistical measure of the average lifespan of a specific population. For advanced age groups, LE is more like the expectant time to death which is affected most strongly by changes in morbidity and health status. Stearns and Norton^{16} indicate that neglecting time to death when considering these issues may be viewed as an omitted variable problem that results in an upward bias on the estimated effect of age. With the time-to-death hypothesis, a shift in the mortality risk to higher ages will not affect lifetime HCE as a death occurs only once in every life.^{17} If most people spend a certain amount of money in their last year, the time to death is more important than calendar age when we examine the determinant of health expenditure. Disregarding the effects of increasing longevity on HCE, we perhaps overestimate the impact of population ageing on future expenditure.
Economic development brings medical progress, a fall in mortality, an increase in chronic disease and a lengthening of normal lifespan. Regarding the health of the elderly, there have been several hypotheses to explain the impact of changes in mortality, morbidity, disability and LE on HCE. In addition to the expansion and compression of morbidity hypotheses, the third hypothesis is the dynamic equilibrium proposed by Manton,^{18} which claims that mortality reductions will be associated with a redistribution of disease and disability. Thus, while the proportion of the lifespan with serious illness or disability stabilises or decreases, the proportion with moderate disability or less severe illness increases. Building on these earlier hypotheses, Michel and Robine^{19} proposed a new theory of the ageing population to suggest how the expression and compression of morbidity may co-exist today and in the future. For health insurance systems and policymakers, it is obviously important to analyse which of these scenarios is unfolding, along with the associated impact on HCE.
In this study, we emphasise the impact of longevity on HCE with an ageing population, aiming to examine whether longer life expectancies will increase or decrease HCE. This study differs from the earlier literature in that it adds factors related to longevity to re-estimate the population demographic effect on HCE. We use the LE at specific ages to proxy the time-to-death variable in order to find the relationship with HCE. First, on the basis of the time-to-death hypothesis, we assume that there possibly exists a negative relationship between LE and HCE. Second, we want to investigate whether the increase in LE would aggravate or mitigate the growth of HCE. If we find a negative impact on HCE, it will imply that the compression of morbidity is happening in Taiwan. Medical expenditure on the senior population may be reduced by their better health status and healthier lifestyles. In contrast with prior studies, we are interested in the correlation between population ageing, LE and HCE. With the use of cointegration analysis, we attempt to estimate the impact of population ageing and LE on HCE in the long term, and to investigate whether there is compression or expansion of morbidity in Taiwan.
Data and methodology
In the previous studies, the main determinants of HCE were personal income, population ageing, technological change and other supply variables of medical services. This paper re-estimates the long-run economic relationship between HCE and related demographic variables, using time-series data in Taiwan during the period 1960–2006. In this data, the HCE and the income proxy, which is gross domestic product (denoted as GDP), are expressed in real per capita terms, with prices expressed in 2001 terms. For the choice of demographic variables, we are concerned that omitting the time-to-death variable will raise the bias on the estimated effect of age,^{20} and that if the LE of the aged population is increasing, this bias would increase over time. For this reason, we take the LE at specific ages as a time-to-death proxy in the aggregated model of HCE.
We thus separate the demographic effect into two aspects, population ageing and LE. With regard to the distribution of the aged population, we use the dependency ratio of the elderly population (denoted as AGE) to be the population ageing proxy, as in Chou.^{21} Following the demographic statistics definition, the aged dependency ratio is equal to the number of individuals aged over 65 divided by the number of individuals aged 15–64, expressed as a percentage. We choose three specific age groups from 65 to 79, with every 5 years classified as one group, to estimate the impact of LE. The LE proxy for the age group 65–69 is denoted as LE65, the LE at the age group 70–74 is denoted as LE70, and the LE at the age group 75–79 is denoted as LE75.
Variables and definitions of regression
Variable | Definition |
---|---|
HCE | Real per capital personal health care expenditures |
GDP | Real per capital gross domestic product |
AGE | Dependency ratio of the old-aged population |
LE65 | Life expectancy for population aged 65–69 years |
LE70 | Life expectancy for population aged 70–74 years |
LE75 | Life expectancy for population aged 75–79 years |
DR | Number of practicing physicians per hundred thousand persons |
We use the Johansen^{25} and Johansen and Juselius^{26} maximum likelihood procedure to capture the long-run quantitative impact of changes in GDP, AGE, LE, LEC, and DR on HCE variation. In the cointegration process, we use two tests, the maximal eigenvalue and trace tests, to determine the number of cointegrating vectors and long-run equilibrium relationships. For the potential endogeneity bias, we use two bias-correction methods, dynamic ordinary least squares (DOLS) and fully modified (FM) OLS, to deal with the endogeneity and serial dependence of regressors, as in Okunade and Murthy^{27} and Chou.^{28}
Our study uses the annual macroeconomic data in Taiwan over the period from 1960 to 2006. The major sources for HCE, GDP, DR and related demographic data are obtained from the Taiwan national statistical information web site database,^{29} and all the variables in our model are expressed in natural logarithmic form.
Empirical results
Most macroeconomic time series seem to exhibit strong trends and so are not stationary and are therefore inappropriate for use in OLS estimation.^{30} In order to avoid spurious regressions and meaningless results, the texts on time series econometrics^{31} have highlighted the importance of testing time-series data for the presence of a unit root. The unit root test results are presented in Table A1 of Appendix 1. We investigate the presence of the unit root in the level variables, HCE, GDP, AGE, LC65, LC70 and LC75. Accounting the unit root test results, we find all variables are obviously non-stationary in level but stationary in first differences, I(1), at a 5 per cent level of significance. We then use the cointegration procedure to examine the long-run relationship between the variables.
Normalised cointegration vector
Variables | Model II^{a} | Model III^{b} | Model VI^{b} | |||
---|---|---|---|---|---|---|
HCE_{t} | 1 | 1 | 1 | |||
GDP_{t} | −0.98 | (−7.06)^{**} | −1.01 | (−7.62)^{**} | −0.97 | (−6.80)^{**} |
AGE_{t} | −0.27 | (−6.15)^{**} | −0.37 | (−7.09)^{**} | −0.39 | (−6.41)^{**} |
LE65_{t} | 6.53 | (4.81)^{**} | ||||
LE70_{t} | 7.04 | (6.02)^{**} | ||||
LE75_{t} | 5.93 | (5.77)^{**} | ||||
DR_{t} | −0.88 | (−3.95)^{**} | −0.61 | (−2.67)^{**} | −0.46 | (−1.72) |
C | −10.17 | −9.41 | −5.36 | |||
Error-correction | −1.18 | (−3.18)^{**} | −0.15 | (−2.09)^{*} | −0.11 | (−2.32)^{*} |
The observed asymptotic t-values are shown in parentheses. We find the error-correction terms (ECT) are highly significant with the expected negative sign, and for equations (5) to (7) are −1.18, −0.15, and −0.11, respectively. Those highly significant and negative ECT support the validity of a long-run equilibrium relationship among HCE, GDP, AGE, DR and the related LE variables. Furthermore, the coefficients of GDP, AGE and DR are significant and positive at the 1 per cent level. These results conform to the past empirical results on the determination of HCE. The estimated coefficient of GDP as the long run income elasticity of health demand is between 0.97 and 1.01, meaning that health care at the aggregate level appears to be a necessity good. We can also find that the impact of population ageing is positive but small in every regression. As regards LE, we observed that the coefficients of LE65, LE70 and LE75 are significant and negative in the long run. The expected remaining life, or the time to death, is the major determinant of health care costs and thus the increase in LE would cause health expenditure to decrease. This implies that we do not need to worry that the expenditure will rise as the people become aged and live longer. On the other hand, the results support the compression of morbidity hypothesis, in that there is a negative relationship between the LE for the three aged groups and HCE. We use the restrictive function to test the coefficient restrictions of the vector error correction model. Based on the likelihood ratio (χ^{2}) exclusion test results, the vector coefficients of AGE, LE65 (or LE70, LE75), and DR are significant and equal to (1, −1, 1), respectively. The observed LR statistics (χ^{2}) of Models II–VI are all smaller than the χ^{2} critical value with two degrees of freedom with a 5 per cent statistical significance level, suggesting that we cannot reject the null hypothesis that the vector coefficients of AGE, LE65 (or LE70, LE75), and DR are significant and equal to (1, −1, 1), respectively, in Models II–IV. From the Johansen cointegration tests and the vector error correction model, we find that HCE, AGE, LE65 (or LE70, LE75) and DR have an equilibrium relationship in the long run.
Using OLS, DOLS, and FMOLS methods to estimate Models II–VI (dependent variable: HCE)
Model | Variables | OLS | DOLS | FMOLS | |||
---|---|---|---|---|---|---|---|
Model II | GDP_{t} | 0.34 | (5.11)^{**} | 0.15 | (3.26)^{**} | 0.21 | (1.71)^{*} |
AGE_{t} | 0.29 | (11.33)^{**} | 0.07 | (2.08)^{*} | 0.27 | (7.09)^{**} | |
LE65_{t} | −0.78 | (−1.11) | −0.91 | (−2.06)^{*} | −0.08 | (−1.14) | |
DR_{t} | 0.01 | (0.10) | 0.20 | (2.52)^{*} | 0.14 | (0.85) | |
C | 1.09 | (0.74) | 1.49 | (2.95)^{**} | 0.03 | (0.07) | |
R-squared | 0.99 | 0.99 | 0.98 | ||||
D.W. | 0.51 | 1.58 | 1.17 | ||||
Model III | GDP_{t} | 0.31 | (5.18)^{**} | 0.12 | (5.18)^{**} | 0.19 | (2.42)^{*} |
AGE_{t} | 0.28 | (9.11)^{**} | 0.06 | (9.11)^{**} | 0.26 | (6.39)^{**} | |
LE65_{t} | −0.26 | (−0.48) | −0.50 | (−2.18)^{*} | −0.24 | (−1.69) | |
DR_{t} | 0.01 | (0.02) | 0.17 | (2.16)^{*} | 0.14 | (1.06) | |
C | 0.01 | (−0.01) | 0.55 | (2.91)^{**} | 0.11 | (1.34) | |
R-squared | 0.99 | 0.99 | 0.98 | ||||
D.W. | 0.48 | 1.52 | 1.25 | ||||
Model VI | GDP_{t} | 0.29 | (5.47)^{**} | 0.11 | (2.65)^{**} | 0.19 | (1.45) |
AGE_{t} | 0.27 | (11.87)^{**} | 0.06 | (1.62)^{**} | 0.25 | (6.39)^{**} | |
LE65_{t} | −0.09 | (−1.92) | −0.32 | (−2.04)^{*} | −0.26 | (−0.76) | |
DR_{t} | 0.01 | (0.05) | 0.17 | (2.24)^{*} | 0.14 | (0.96) | |
C | −0.35 | (1.42) | 0.15 | (0.32) | −0.10 | (−0.47) | |
R-squared | 0.99 | 0.99 | 0.97 | ||||
D.W. | 0.47 | 1.51 | 1.26 |
Conclusion and policy implications
In this paper we re-estimate the demographic impact on HCE during the period of 1960–2006 in Taiwan. In addition to population ageing, we especially add LE from advanced ages to examine the demographic impact on HCE. The results are robust to a battery of unit root and cointegration tests, and we find that HCE, income, population ageing, LE and the number of practicing physicians have statistic significance and long-term economic relationship in Taiwan. Our data shows population ageing, proxied by the dependency ratio, has a significantly positive but weak impact on HCE. The LE at the age groups of 65, 70, and 75 has a significantly negative impact on HCE and the coefficient becomes smaller as the age level rises. Our results thus support the compression of morbidity hypothesis. Longevity and population ageing do not inevitably lead to an increase in HCE. People possibly have more healthy LE at the advanced ages.
The conventional wisdom is that health care costs increase with an ageing population. However, this ageing was not found to be a significant factor in explaining the variations of HCE in prior research,^{37} although this might be due to the problem of omitting a variable. After taking into account LE at various advanced ages, we find that population ageing, proxied by the dependency ratio, does have a positive impact on HCE. This finding is consistent with Chou.^{38}
Consistent with the compression of morbidity hypothesis, our findings indicate that if people live longer and more healthily then the effect of ageing on health costs may be offset by that of LE, which is likely to have meaningful implications for policymaking and future research. If the extension of LE is due to good life style and health status, the authorities concerned should consider enhancing public education to improve health, and so we do not necessarily need to worry about the adverse impact on health expenditure as LE increases. One of the limitations of this study is that LE represents the length of life prior to death, but does not measure the quality of life and health status. The World Health Organization proposed a general model of health transition to evaluate the consequences of an increase in survival of health status in 1984.^{39} This model calculates not only ordinary LE, but also disease-free/disability-free LE.^{40} Future research could use the disability-adjusted LE data to more accurately estimate the impact of healthy LE on HCE. On the other hand, different diseases may have different effects on the morbidity compression, and future research should use individual disease data to investigate the existence of morbidity compression in Taiwan.
Hitiris and Posnett (1992); Hansen and King (1996); Barros (1998); Gerdtham and Lothgren (2000); Di Matteo (2005); Martins et al. (2006).
Zweifel et al. (1999, 2004); Seshamani and Gray (2004); Stearns and Norton (2004); Dormont et al. (2006).
Hodgson (1992); Daviglus et al. (1998); Spillman and Lubitz (2000); Miller (2001); Lubitz et al. (2003).
See, for example, Newhouse (1977); Gerdtham et al. (1992); Hitiris and Posnett (1992); Zweifel et al. (2004).