Journal of Behavioral Medicine

, Volume 23, Issue 5, pp 437–450

Chest Pain and the Treatment of Psychosocial/Emotional Distress in CAD Patients

Authors

  • Mark W. Ketterer
    • Department of Psychiatry and Heart and Vascular Institute, Henry Ford Health Sciences CenterCase Western Reserve University
    • Henry Ford Hospital/CFP3
  • Faye Fitzgerald
    • Department of Psychiatry and Heart and Vascular Institute, Henry Ford Health Sciences CenterCase Western Reserve University
  • Steve Keteyian
    • Department of Psychiatry and Heart and Vascular Institute, Henry Ford Health Sciences CenterCase Western Reserve University
  • Beth Thayer
    • Department of Psychiatry and Heart and Vascular Institute, Henry Ford Health Sciences CenterCase Western Reserve University
  • Margaret Jordon
    • Department of Psychiatry and Heart and Vascular Institute, Henry Ford Health Sciences CenterCase Western Reserve University
  • Cathy McGowan
    • Department of Psychiatry and Heart and Vascular Institute, Henry Ford Health Sciences CenterCase Western Reserve University
  • Greg Mahr
    • Department of Psychiatry and Heart and Vascular Institute, Henry Ford Health Sciences CenterCase Western Reserve University
  • Antoun Manganas
    • Department of Psychiatry and Heart and Vascular Institute, Henry Ford Health Sciences CenterCase Western Reserve University
  • A. David Goldberg
    • Department of Psychiatry and Heart and Vascular Institute, Henry Ford Health Sciences CenterCase Western Reserve University
Article

DOI: 10.1023/A:1005521014919

Cite this article as:
Ketterer, M.W., Fitzgerald, F., Keteyian, S. et al. J Behav Med (2000) 23: 437. doi:10.1023/A:1005521014919

Abstract

Treatment of psychosocial/emotional distress as a strategy for diminishing chest pain in such patients remains entirely unutilized in standard care. Sixty-three patients with known or suspected CAD were entered in an aggressive lifestyle modification program. Patients completed the Symptom Checklist 90—Revised (SCL90R) at the diagnostic interview session, at 3 and at 12 months. Statistically significant drops were observed on multiple scales of the SCL90R at both 3 and 12 months. An item from the SCL90R was used as a proxy for angina. Multiple measures of emotional distress at baseline were found to correlate with chest pain at baseline, but not a number of traditional cardiovascular risk factors. The chest pain item displayed improvement at both 3 and 12 months. Improvement on all scales of the SCL90R correlated with improvement in chest pain. It may be possible to control chest pain in some CAD patients with psychosocial interventions.

ischemic heart diseaseanginastroke

Copyright information

© Plenum Publishing Corporation 2000