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Unstructured Socializing with Peers and Delinquent Behavior: A Genetically Informed Analysis

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Abstract

A large body of research finds that unstructured socializing with peers is positively associated with delinquency during adolescence. Yet, existing research has not ruled out the potential for confounding due to genetic factors and factors that can be traced to environments shared between siblings. To fill this void, the current study examines whether the association between unstructured socializing with peers and delinquent behavior remains when accounting for genetic factors, shared environmental influences, and a variety of non-shared environmental covariates. We do so by using data from the twin subsample of the National Longitudinal Study of Adolescent to Adult Health (n = 1200 at wave 1 and 1103 at wave 2; 51% male; mean age at wave 1 = 15.63). Results from both cross-sectional and lagged models indicate the association between unstructured socializing with peers and delinquent behavior remains when controlling for both genetic and environmental influences. Supplementary analyses examining the association under different specifications offer additional, albeit qualified, evidence supportive of this finding. The study concludes with a discussion highlighting the importance of limiting free time with friends in the absence of authority figures as a strategy for reducing delinquency during adolescence.

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Notes

  1. We drew responses from the in-home survey even though this question was asked during the in-school survey as well. We opted to use the in-home survey version because it was only during that phase of the Add Health study design that researchers made an effort to over-sample twins. Thus, focusing on responses to the in-home survey will give us the best chances to preserve sample sizes in light of missing data concerns.

  2. We would have preferred to control for peer delinquency, as opposed to peer substance use. Unfortunately, two things precluded us from doing so for this study. First, respondent reports of the delinquent behavior of their friends are not included as part of the design of the Add Health study. Second, while past research has made use of the social-networking measure of peer delinquency (e.g., Haynie and Osgood 2005) contained within the Add Health data as a control variable in models assessing the association between unstructured socializing and delinquency, the network data was not obtained for all Add Health participants. In conjunction with the restricted focus on twins from the Add Health, the inclusion of the social-networking measure of peer delinquency would have cut our analytic sample sizes, which already are not very large, in half. Given these circumstances, we feel that the use of the perceptual measure of peer substance use is a reasonable proxy measure for peer delinquency, albeit imperfect.

  3. We also re-estimated the DF model after restricting it to cases that were included in the regression models presented in Tables 3 and 4; results were substantively unchanged.

  4. Prior research using the Add Health data establishes that delinquency is under heritable influence. Given this, we elected not to estimate DF models for delinquency because it would be redundant. Readers interested in information on the heritability of delinquency using the Add Health data are referred to such studies as Boisvert et al. (2014) and Wright et al. (2008).

  5. We thank an anonymous reviewer for this suggestion.

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Acknowledgements

The authors wish to thank Evelien Hoeben for helpful comments on earlier drafts of the manuscript. This study uses data from Add Health, a program project directed by Kathleen Mullan Harris and designed by J. Richard Udry, Peter S. Bearman, and Kathleen Mullan Harris at the University of North Carolina at Chapel Hill, and funded by grant P01-HD31921 from the Eunice Kennedy Shriver National Institute of Child Health and Human Development, with cooperative funding from 23 other federal agencies and foundations. Special acknowledgment is due Ronald R. Rindfuss and Barbara Entwisle for assistance in the original design. Information on how to obtain the Add Health data files is available on the Add Health website (http://www.cpc.unc.edu/addhealth).

Funding

No direct support was received from grant P01-HD31921 for the current study.

Author Contributions

R.C.M. conceived of the study, drafted the introduction and literature review sections of the manuscript, and drafted portions of the discussion section; J.C.B. acquired the data for the analysis, conducted the statistical analysis, drafted portions of the methods, results, and discussion sections, and created the tables. All authors read, edited, and approved the final manuscript.

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Correspondence to Ryan C. Meldrum.

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Conflict of Interest

The authors declare that they have no competing interests.

Ethical Approval

The procurement of the data required for this study was approved by the Institutional Review Board of the University of Cincinnati.

Informed Consent

Consent for participation in the Add Health study was obtained from both the parents of the participants and the participants themselves at the time the study began in the 1990s.

Appendices

Appendix 1. Information on DF Model Estimation

The DF model takes the following form:

$${X_{1j}} = {\beta _0} + {\beta _1}{\ddot X_{2j}} + {\beta _2}{\ddot X_{2j}}*{R_j} + {\varepsilon _{1j}}$$

where: X 1j is the score on unstructured socializing for twin 1 from twin pair j; β 0 is the intercept and is typically not interpreted substantively; \({\beta _1}{\ddot X_{2j}}\) provides an estimate of the shared environment by estimating the impact of twin 2’s mean-centered score of X on his/her co-twin’s score; \({\beta _2}{\ddot X_{2j}}*{R_j}\) provides an estimate of heritability by calculating the degree to which the mean-centered version of X 2 is a better predictor of X 1 for siblings that share more genetic overlap (i.e., when R = 1 for MZ twins relative to R = 0.50 for DZ twins); and ε 1j is the error term that captures all sources of variance in X 1j that are not attributable to heritability or shared environments. Note that we estimated the DF model using double-entry, which required us to adjust the standard errors to account for the clustering of twins in pairs.

Appendix 2. Information on Fixed Effects Model Estimation

The fixed effects model takes the following form:

$${Y_{ij}} = {\varphi _0} + {\varphi _1}{X_{ij}} + {\varphi _2}{D_{1j}} + \mathop {\sum}\limits_{j = 1}^J {{\varphi _j}}$$

where: Y ij is the outcome of focus; φ 0 is an intercept term revealing the average level of Y for respondents who score a zero on all the right-hand side variables; φ 1 X ij captures the impact of unstructured socializing on Y; φ 2 D 1j is a fixed effect that captures any random differences that may exist between twins labeled twin 1 and those labeled twin 2; and \(\mathop {\sum}\nolimits_{j = 1}^J {{\varphi _{j\,}}}\) captures the collective influence of all the twin-level fixed effects for all twin pairs J.

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Meldrum, R.C., Barnes, J.C. Unstructured Socializing with Peers and Delinquent Behavior: A Genetically Informed Analysis. J Youth Adolescence 46, 1968–1981 (2017). https://doi.org/10.1007/s10964-017-0680-x

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