Abstract
A large body of research finds that unstructured socializing with peers is positively associated with delinquency during adolescence. Yet, existing research has not ruled out the potential for confounding due to genetic factors and factors that can be traced to environments shared between siblings. To fill this void, the current study examines whether the association between unstructured socializing with peers and delinquent behavior remains when accounting for genetic factors, shared environmental influences, and a variety of non-shared environmental covariates. We do so by using data from the twin subsample of the National Longitudinal Study of Adolescent to Adult Health (n = 1200 at wave 1 and 1103 at wave 2; 51% male; mean age at wave 1 = 15.63). Results from both cross-sectional and lagged models indicate the association between unstructured socializing with peers and delinquent behavior remains when controlling for both genetic and environmental influences. Supplementary analyses examining the association under different specifications offer additional, albeit qualified, evidence supportive of this finding. The study concludes with a discussion highlighting the importance of limiting free time with friends in the absence of authority figures as a strategy for reducing delinquency during adolescence.
Similar content being viewed by others
Notes
We drew responses from the in-home survey even though this question was asked during the in-school survey as well. We opted to use the in-home survey version because it was only during that phase of the Add Health study design that researchers made an effort to over-sample twins. Thus, focusing on responses to the in-home survey will give us the best chances to preserve sample sizes in light of missing data concerns.
We would have preferred to control for peer delinquency, as opposed to peer substance use. Unfortunately, two things precluded us from doing so for this study. First, respondent reports of the delinquent behavior of their friends are not included as part of the design of the Add Health study. Second, while past research has made use of the social-networking measure of peer delinquency (e.g., Haynie and Osgood 2005) contained within the Add Health data as a control variable in models assessing the association between unstructured socializing and delinquency, the network data was not obtained for all Add Health participants. In conjunction with the restricted focus on twins from the Add Health, the inclusion of the social-networking measure of peer delinquency would have cut our analytic sample sizes, which already are not very large, in half. Given these circumstances, we feel that the use of the perceptual measure of peer substance use is a reasonable proxy measure for peer delinquency, albeit imperfect.
Prior research using the Add Health data establishes that delinquency is under heritable influence. Given this, we elected not to estimate DF models for delinquency because it would be redundant. Readers interested in information on the heritability of delinquency using the Add Health data are referred to such studies as Boisvert et al. (2014) and Wright et al. (2008).
We thank an anonymous reviewer for this suggestion.
References
Anderson, A. L., & Hughes, L. A. (2009). Exposure to situations conducive to delinquent behavior: The effects of time use, income, and transportation. Journal of Research in Crime and Delinquency, 46, 5–34.
Augustyn, M. B., & McGloin, J. M. (2013). The risk of informal socializing with peers: Considering gender differences across predatory delinquency and substance use. Justice Quarterly, 30, 117–143.
Barnes, G. M., Hoffman, J. H., Welte, J. W., Farrell, M. P., & Dintcheff, B. A. (2007). Adolescents’ time use: Effects on substance use, delinquency and sexual activity. Journal of Youth and Adolescence, 36, 697–710.
Barnes, J. C. (2013). Analyzing the origins of life-course-persistent offending: A consideration of environmental and genetic influences. Criminal Justice and Behavior, 40, 519–540.
Barnes, J. C., & Boutwell, B. B. (2013). A demonstration of the generalizability of twin-based research on antisocial behavior. Behavior Genetics, 43, 120–131.
Barnes, J. C., Boutwell, B. B., Beaver, K. M., Gibson, C. L., & Wright, J. P. (2014). On the consequences of ignoring genetic influences in criminological research. Journal of Criminal Justice, 42, 471–482.
Barnes, J. C., & Meldrum, R. C. (2015). The impact of sleep duration on adolescent development: A genetically informed analysis of identical twin pairs. Journal of Youth and Adolescence, 44, 489–506.
Beaver, Kevin M. (2008). Nonshared environmental influence on adolescent delinquent involvement and adult criminal behavior. Criminology, 46, 341–369.
Boisvert, D., Boutwell, B. B., Vaske, J., & Newsome, J. (2014). Genetic and environmental overlap between delinquent peer association and delinquency in adolescence. Criminal Justice and Behavior, 41, 58–74.
Cohen, L. E., Cantor, D., & Kluegel, J. R. (1981). Robbery victimization in the United States: An analysis of a nonrandom event. Social Science Quarterly, 62, 644–657.
Cohen, L. E., & Felson, M. (1979). Social change and crime rate trends: A routine activity approach. American Sociological Review, 44, 588–608.
DeFries, J. C., & Fulker, D. W. (1985). Multiple regression analysis of twin data. Behavior Genetics, 15, 467–473.
Dipietro, S. M., & McGloin, J. M. (2012). Differential susceptibility? Immigrant youth and peer influence. Criminology, 50, 711–742.
Eaves, L., & Eysenck, H. (1975). The nature of extraversion: A genetical analysis. Journal of Personality and Social Psychology, 32, 102–112.
Felson, R. B., Osgood, W. D., Horney, J., & Wiernik, C. (2012). Having a bad month: General versus specific effects of stress on crime. Journal of Quantitative Criminology, 28, 347–363.
Fleming, C. B., Catalano, R. F., Mazza, J. J., Brown, E. C., Haggerty, K. P., & Harachi, T. W. (2008). After-school activities, misbehavior in school, and delinquency from the end of elementary school through the beginning of high school: A test of social development model hypotheses. The Journal of Early Adolescence, 28, 277–303.
Goldstein, S. E., Davis-Kean, P. E., & Eccles, J. S. (2005). Parents, peers, and problem behavior: A longitudinal investigation of the impact of relationship perceptions and characteristics on the development of adolescent problem behavior. Developmental Psychology, 41, 401–413.
Greene, K., & Banerjee, S. C. (2009). Examining unsupervised time with peers and the role of association with delinquent peers on adolescent smoking. Nicotine & Tobacco Research, 11, 371–380.
Harris, K. M. (2009). The national longitudinal study of adolescent health (Add Health), Waves I & II, 1994–1996; Wave III, 2001–2002; Wave IV, 2007–2009 [machine-readable data file and documentation]. Chapel Hill, NC: Carolina Population Center, University of North Carolina at Chapel Hill.
Harris, K. M., Halpern, C. T., Whitsel, E., Hussey, J., Tabor, J., Entzel, P., & Udry, J. R. (2009). The national longitudinal study of adolescent health: Research design. http://www.cpc.unc.edu/projects/addhealth/design.
Haynie, D. L., & Osgood, D. W. (2005). Reconsidering peers and delinquency: How do peers matter? Social Forces, 84, 1109–1130.
Higgins, G. E., & Jennings, W. G. (2010). Is unstructured socializing a dynamic process? An exploratory analysis using a semiparametric group-based modeling approach. Criminal Justice Review, 35, 514–532.
Hoeben, E. M., Meldrum, R. C., Walker, D., & Young, Y. T. N. (2016). The role of peer delinquency and unstructured socializing in explaining delinquency and substance use: A state-of-the-art review. Journal of Criminal Justice, 47, 108–122.
Hoeben, E. M., & Weerman, F. M. (2014). Situational conditions and adolescent offending: Does the impact of unstructured socializing depend on its location? European Journal of Criminology, 11, 481–499.
Jacobson, K. C., & Rowe, D. C. (1998). Genetic and shared environmental influences on adolescent BMI: Interactions with race and sex. Behavior Genetics, 28, 265–278.
Janssen, H. J., Weerman, F. M., & Eichelsheim, V. I. (2017). Parenting as a protective factor against criminogenic settings? Interaction effects between three aspects of parenting and unstructured socializing in disordered areas. Journal of Research in Crime and Delinquency, 54, 181–207.
Kennedy, L. W., & Forde, D. R. (1990). Routine activities and crime: An analysis of victimization in Canada. Criminology, 28, 137–152.
Kohler, H., Behrman, J. R., & Schnittker, J. (2011). Social science methods for twins data: Integrating causality, endowments, and heritability. Biodemography and Social Biology, 57, 88–141.
Lotz, R., & Lee, L. (1999). Sociability, school experience, and delinquency. Youth & Society, 31, 199–223.
Maimon, D., & Browning, C. R. (2010). Unstructured socializing, collective efficacy, and violent behavior among urban youth. Criminology, 48, 443–474.
Meldrum, R. C., Barnes, J. C., & Hay, C. (2015). Sleep deprivation, low self-control, and delinquency: A test of the strength model of self-control. Journal of Youth and Adolescence, 44, 465–477.
Meldrum, R. C., & Clark, J. (2015). Adolescent virtual time spent socializing with peers, substance use, and delinquency. Crime & Delinquency, 61, 1104–1126.
Meldrum, R. C., Young, J. T. N., & Weerman, F. M. (2009). Reconsidering the effect of self-control and delinquent peers: Implications of measurement for theoretical significance. Journal of Research in Crime and Delinquency, 46, 353–376.
Messner, S. F., & Tardiff, K. (1985). The social ecology of urban homicide: An application of the “routine activities” approach. Criminology, 23, 241–267.
Miller, J. (2013). Individual offending, routine activities, and activity settings: Revisiting the routine activity theory of general deviance. Journal of Research in Crime and Delinquency, 50, 390–416.
Osgood, D. W., & Anderson, A. L. (2004). Unstructured socializing and rates of delinquency. Criminology, 42, 519–550.
Osgood, D. W., Wilson, J. K., O’Malley, P. M., Bachman, J. G., & Johnston, L. D. (1996). Routine activities and individual deviant behavior. American Sociological Review, 61, 635–655.
Plomin, R., & Daniels, D. (1986). Genetics and shyness. In W. Jones, J. Cheek, and S. Briggs (Eds.), Shyness: Perspectives on Research and Treatment (pp. 63–80). New York: Springer.
Plomin, R., DeFries, J. C., Knopik, V. S., & Neiderheiser, J. (2013). Behavioral Genetics (6th Edn). New York, NY: Worth Publishers.
Polderman, T. J., Benyamin, B., De Leeuw, C. A., Sullivan, P. F., Van Bochoven, A., Visscher, P. M., & Posthuma, D. (2015). Meta-analysis of the heritability of human traits based on fifty years of twin studies. Nature Genetics, 47, 702–709.
Raine, A. (2013). The anatomy of violence: The biological roots of crime. New York, NY: Pantheon.
Rodgers, J. L., & Kohler, H. (2005). Reformulating and simplifying the DF analysis model. Behavior Genetics, 35, 211–217.
Rodgers, J. L., Buster, M., & Rowe, D. C. (2001). Genetic and environmental influences on delinquency: DF analysis of NLSY kinship data. Journal of Quantitative Criminology, 17, 145–168.
Taylor, J., Iacono, W. G., & McGue, M. (2000). Evidence for a genetic etiology of early-onset delinquency. Journal of Abnormal Psychology, 109(4), 634–643.
TenEyck, M., & Barnes, J. C. (2015). Examining the impact of peer group selection on self-reported delinquency: A consideration of active gene–environment correlation. Criminal Justice and Behavior, 42, 741–762.
Thomas, K. J., & McGloin, J. M. (2013). A dual‐systems approach for understanding differential susceptibility to processes of peer influence. Criminology, 51, 435–474.
Turkheimer, E., & Harden, K. P. (2013). Behavior genetic research methods: Testing quasi-causal hypotheses using multivariate twin data. In H. T. Reis, C. M. Judd (Eds.), Handbook of research methods in social and personality psychology. 2nd edn. New York, NY: Cambridge University Press.
Van den Berg, S. M., De Moor, M. H., McGue, M., Pettersson, E., Terracciano, A., Verweij, K. J., et al. (2014). Harmonization of neuroticism and extraversion phenotypes across inventories and cohorts in the genetics of personality consortium: An application of item response theory. Behavior Genetics, 44, 295–313.
Weerman, F. M., Wilcox, P., & Sullivan, C. J. (2017). The short-term dynamics of peers and delinquent behavior: An analysis of bi-weekly changes within a high school student network. Journal of Quantitative Criminology, Onlinefirst Edition.
Weerman, F. M., Bernasco, W., Bruinsma, G. J., & Pauwels, L. J. (2016). Gender differences in delinquency and situational action theory: A partial test. Justice Quarterly, 33, 1182–1209.
Weerman, F. M., Bernasco, W., Bruinsma, G. J., & Pauwels, L. J. (2015). When is spending time with peers related to delinquency? The importance of where, what, and with whom. Crime & Delinquency, 61, 1386–1413.
Wright, J. P., & Beaver, K. M. (2005). Do parents matter in creating self‐control in their children? A genetically informed test of Gottfredson and Hirschi’s theory of low self‐control. Criminology, 43, 1169–1202.
Wright, J. P., Beaver, K. M., Delisi, M., & Vaughn, M. (2008). Evidence of negligible parenting influences on self‐control, delinquent peers, and delinquency in a sample of twins. Justice Quarterly, 25, 544–569.
Wright, K. A., Kim, B., Chassin, L., Losoya, S. H., & Piquero, A. R. (2014). Ecological context, concentrated disadvantage, and youth reoffending: Identifying the social mechanisms in a sample of serious adolescent offenders. Journal of Youth and Adolescence, 43, 1781–1799.
Acknowledgements
The authors wish to thank Evelien Hoeben for helpful comments on earlier drafts of the manuscript. This study uses data from Add Health, a program project directed by Kathleen Mullan Harris and designed by J. Richard Udry, Peter S. Bearman, and Kathleen Mullan Harris at the University of North Carolina at Chapel Hill, and funded by grant P01-HD31921 from the Eunice Kennedy Shriver National Institute of Child Health and Human Development, with cooperative funding from 23 other federal agencies and foundations. Special acknowledgment is due Ronald R. Rindfuss and Barbara Entwisle for assistance in the original design. Information on how to obtain the Add Health data files is available on the Add Health website (http://www.cpc.unc.edu/addhealth).
Funding
No direct support was received from grant P01-HD31921 for the current study.
Author Contributions
R.C.M. conceived of the study, drafted the introduction and literature review sections of the manuscript, and drafted portions of the discussion section; J.C.B. acquired the data for the analysis, conducted the statistical analysis, drafted portions of the methods, results, and discussion sections, and created the tables. All authors read, edited, and approved the final manuscript.
Author information
Authors and Affiliations
Corresponding author
Ethics declarations
Conflict of Interest
The authors declare that they have no competing interests.
Ethical Approval
The procurement of the data required for this study was approved by the Institutional Review Board of the University of Cincinnati.
Informed Consent
Consent for participation in the Add Health study was obtained from both the parents of the participants and the participants themselves at the time the study began in the 1990s.
Appendices
Appendix 1. Information on DF Model Estimation
The DF model takes the following form:
where: X 1j is the score on unstructured socializing for twin 1 from twin pair j; β 0 is the intercept and is typically not interpreted substantively; \({\beta _1}{\ddot X_{2j}}\) provides an estimate of the shared environment by estimating the impact of twin 2’s mean-centered score of X on his/her co-twin’s score; \({\beta _2}{\ddot X_{2j}}*{R_j}\) provides an estimate of heritability by calculating the degree to which the mean-centered version of X 2 is a better predictor of X 1 for siblings that share more genetic overlap (i.e., when R = 1 for MZ twins relative to R = 0.50 for DZ twins); and ε 1j is the error term that captures all sources of variance in X 1j that are not attributable to heritability or shared environments. Note that we estimated the DF model using double-entry, which required us to adjust the standard errors to account for the clustering of twins in pairs.
Appendix 2. Information on Fixed Effects Model Estimation
The fixed effects model takes the following form:
where: Y ij is the outcome of focus; φ 0 is an intercept term revealing the average level of Y for respondents who score a zero on all the right-hand side variables; φ 1 X ij captures the impact of unstructured socializing on Y; φ 2 D 1j is a fixed effect that captures any random differences that may exist between twins labeled twin 1 and those labeled twin 2; and \(\mathop {\sum}\nolimits_{j = 1}^J {{\varphi _{j\,}}}\) captures the collective influence of all the twin-level fixed effects for all twin pairs J.
Rights and permissions
About this article
Cite this article
Meldrum, R.C., Barnes, J.C. Unstructured Socializing with Peers and Delinquent Behavior: A Genetically Informed Analysis. J Youth Adolescence 46, 1968–1981 (2017). https://doi.org/10.1007/s10964-017-0680-x
Received:
Accepted:
Published:
Issue Date:
DOI: https://doi.org/10.1007/s10964-017-0680-x