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Septic shock-related acute esophageal necrosis and stenosis: three cases of acquired esophageal stenosis presenting a similar clinical course

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Abstract

We present a case of acute esophageal necrosis (AEN) and two acquired esophageal stenosis cases which showed a similar clinical course after an episode of septic shock. Extensive stenosis of the distal esophagus developed in all cases, which were refractory to dilation therapy and required surgical intervention. The etiology of the latter two cases was deduced from the stenosis after septic shock-induced AEN. Since the diagnosis of AEN is based on its characteristic endoscopic findings which can only be confirmed at the onset of the disease, we therefore called these cases “septic shock-related esophageal stenosis” or abbreviated to “septic esophageal stenosis”. Further study of similar cases is required for understanding the etiology and management of AEN and the relevant disorder which may cause esophageal stenosis.

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Ethical Statement

This article does not contain any studies with human or animal subjects performed by any of the authors.

Conflict of interest

The authors declare that they have no conflict of interest.

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Authors and Affiliations

  1. Department of Gastroenterological Surgery II, Hokkaido University Graduate School of Medicine, West-7, North-15, Kitaku, Sapporo, Hokkaido, 060-8638, Japan

    Toshiaki Shichinohe, Yuma Ebihara, Soichi Murakami, Yo Kurashima, Taro Kuramae, Motoshi Kanai, Joe Matsumoto, Takahiro Tsuchikawa & Satoshi Hirano

  2. Department of Surgery, Otaru Kyokai Hospital, Otaru, Hokkaido, 047-0261, Japan

    Masanobu Kusano

Authors
  1. Toshiaki Shichinohe
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  2. Yuma Ebihara
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  3. Soichi Murakami
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  4. Yo Kurashima
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  5. Taro Kuramae
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  6. Motoshi Kanai
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  7. Joe Matsumoto
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  8. Takahiro Tsuchikawa
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  9. Masanobu Kusano
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  10. Satoshi Hirano
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Correspondence to Toshiaki Shichinohe.

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Shichinohe, T., Ebihara, Y., Murakami, S. et al. Septic shock-related acute esophageal necrosis and stenosis: three cases of acquired esophageal stenosis presenting a similar clinical course. Esophagus 12, 304–308 (2015). https://doi.org/10.1007/s10388-014-0447-3

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  • DOI: https://doi.org/10.1007/s10388-014-0447-3

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