ASHENDEL, C.L. (1985). “The phorbol ester receptor: a phospholipid-regulated protein kinase”. Biochem. Biophys. Acta 822: 219–242.
BUFF, K., BRUNDL, A., and BERNDT, J. (1982). “Differential effects of environmental chemicals on liposomal bilayers fluorescence polarization and pesticide-lipid association studies”. Biochim. Biophys. Acta 688: 92–100.
BYCZKOWSKI, J.Z. (1977). “Biochemical and ultrastructural changes in biological membranes induces byp,p′-DDT (chlorophenotanum) and its metabolites”. Pol. J. Pharmacol. Pharm. 29: 411–417.
CABRAL, J.R.P., and GALENDO, D. (1990). “Carcinogenicity study of the pesticide fenvalerate in mice.” Cancer Lett. 49: 13–18.
ENOMOTO, T., MARTEL, N., KANNO, Y., and YAMASAKI, H. (1984). “Inhibition of cell communication between Balb/c3T3 cells by tumor promoters and protection by cAMP.” J. Cell. Physiol. 121: 323–333.
FAO/WHO (1980). 1979 Evaluations of Some Pesticide Residues in Food. Rome, Food and Agriculture Organization of the United Nations (FAO plant production and protection paper 20 suppl.).
FAO/WHO (1981). 1980 Evaluations of some Pesticide Residues in Food. Rome, Food and Agriculture Organization of the United Nations (FAO plant production and protection paper 26 suppl.).
FLODSTROM, S., WARNGARD, L., LJUNGQUIST, S., and AHLBORG, U.G. (1988). “Inhibition of metabolic cooperationin vitro and enhancement of enzyme altered foci incidence in rat liver by the pyrethroid insecticide fenvalerate.” Arch. Toxicol. 61: 218–223.
JONES, O.T., and LEE, A.G. (1986). “Effects of pyrethroids on the activity of a purified (Ca2+−Mg2+)-AT-Pase.” Pesticide Biochem. Physiol. 25: 420–430.
KRUTOVSKIKH, V.A., OYAMADA, M., and YAMASAKI, H. (1991). “Sequential changes of gap-junctional intercellular communications during multistage rat liver carcinogenesis: direct measurement of communicationin vivo.” Carcinogenesis 12: 1701–1706.
MADHUKAR, B.V., YONEYAMA, M., MATSUMURA, F., TROSKO, J.E., and TSUSHIMOTO, G. (1983). “Alteration of calcium transport by tumor promoters, 12-O-tetradecanoyl phorbol-13-acetate andp,p′-dichlorodiphenyltrichloroethane, in the Chinese hamster V79 fibroblast cell line.” Cancer Lett. 18: 251–259.
MESNIL, M., FITZGERALD, D.J., and YAMASAKI, H. (1988). “Phenobarbital specifically reduces gap junction protein mRNA level in rat liver.” Molec. Carcinogen. 1: 79–81.
NEVEU, M.J., HULLY, J.R., PAUL, D.L., and PITOT, H.C.. (1990). “Reversible alteration in the expression of the gap junctional protein connexin 32 during tumor promotion in rat liver and its role during cell proliferation.” Cancer Commun. 2: 21–31.
OHKAWA, H., KANEKO, H., TSUJI, H., and MIYAMOTO, J. (1979). “Metabolism of fenvalerate (Sumicidin) in rats.” J. Pesticide Sci. 4: 143–155.
PARKER, C.M., McCULLOUGH, C.B., GELLATLY, J.B., and JOHNSTON, C.D. (1983). “Toxicologic and carcinogenic evaluation of fenvalerate in the B6C3F1 mouse.” Fund. Appl. Toxicol. 3: 114–120.
PARKER, C.M., PATTERSON, D.R., VAN GELDER, G.A., GORDON, E.B., VALERIO, M.G., and HALL, W.C. (1984). “Chronic toxicity and carcinogenicity evaluation of fenvalerate in rats.” J. Toxicol. Environ. Health 13: 83–97.
RASHATWAR, S.S., and MATSUMURA, F. (1985). “Interaction of DDT and pyrethroids with calmodulin and its significance in the expression of enzyme activities of phosphodiesterase.” Biochem. Pharmacol. 34: 1689–1694.
SELTZER, K.J., and GORDON, M.A. (1986). “Effect of alterations in lipid packing order by hydrophobic colutes on the association state of protein assemblies in model membranes.” Biophys. Chem. 23: 173–181.
SUGIE, S., MORI, H., and TAKAHASHI, M. (1987). “Effect ofin vivo exposure to the liver tumor promoters phenobarbital or DDT on the gap junctions of rat hepatocytes: a quantitative freeze-fracture analysis.” Carcinogenesis 8: 45–51.
TELANG, S., TONG, C., and WILLIAMS, G.M. (1982). “Epigenetic membrane effects of a possible tumor promoting type on cultured liver cells by the non-genotoxic organochlorine pesticides chlorodane and heptachlor.” Carcinogenesis 3: 1175–1178.
TONG, C.C., and WILLIAMS, G.M. (1987). “The effect of tumor promoters on cell-to-cell communication in liver epithelial cell systems.” In: Biochemical Mechanisms and Regulation of Intercellular Communication (H.A. Milman and E. Elmore, eds.). Princeton Scientific Pub. Co., Princeton, NJ. pp. 251–263.
TROSKO, J.E., YOTTI, L.P., WARREN, S.T., TSUSHIMOTO, G., and CHANG, C. (1982). “Inhibition of cell-cell communication by tumor promoters.” In Hecker, E., Fusenig, N.E., Kunz, W., Marks, F., and Thielmann, H.W. (eds.), Carcinogenesis, vol. 7, Cocarcinogenesis and Biological Effects of Tumor Promoters. Raven Press, New York, pp. 565–585
WÄRNGÅRD, L., FRANSSON, R., DRAKENBERG, T.B., FLODSTROM, S., and AHLBORG, U.G., (1988). “Calmodulin involvement in TPA and DDT induced inhibition of intercellular communication.” Chem. Biol. Interact. 65: 41–49.
WILLIAMS, G.M. (1981). “Liver carcinogenesis: the role for some chemicals of an epigenetic mechanism of liver tumor promotion including modification of the cell membrane.” Fd. Cosmet. Toxicol. 19: 577–583.
YAMASAKI, H. (1990). C. “Gap junctional intercellular communication and carcinogenesis.” Carcinogenesis 11: 1051–1058.
ZEILMAKER, M.J., and YAMASAKI, H. (1986). “Inhibition of junctional intercellular communication as a possible short-term test to detect tumor-promoting agents: results with nine chemicals tested by dye transfer assay in Chinese hamster V79 cells.” Cancer Res. 46: 6180–6186.